Characteristics of vaginal smears with amenorrhea

Before reading we encourage you to read: "The criteria for assessing the state of the vagina"

Amenorrhea, or absence of menstrual periods in women, It developed on the basis of dysfunction hypothalamic centers, caused various pathological processes (brain tumor, transferred inflammation, the influence of psychogenic factors, hypothalamic form of obesity and so on.), or as a result of menstrual disorders of peripheral origin (diseases of the uterus, ovary).

Hypothalamic amenorrhea genesis

Hypothalamic amenorrhea genesis most frequently observed. It can proceed both with normal urinary gonadotropins, and with a significant decrease in its. In this and in another case there is no cyclical nature releasing hormone.

When hypothalamic amenorrhea form lutropin selection is often very little or no, follitropin excreted in the same amount, As in the early follicular phase of the cycle. Isolation is normally their estrogen levels in early proliferative phase of the normal menstrual cycle. Excretion pregnandiol sharply reduced.

Colpocytologic especially with amenorrhea estimated by some researchers as either hyperestrogenism in severe proliferation, or as gipoestrogenizm atrophy.

The most common form in the hypothalamic amenorrhea have low monotone degree of proliferation or moderate atrophy. Rarely marked proliferation of the vaginal epithelium, corresponding to the level of EI and CI the average follicular phase of the normal menstrual cycle.

Amenorrhea peripheral origin

Ovarian menstrual irregularities

Ovarian menstrual irregularities, lead to amenorrhea, or opsomenoree (lengthening the pauses between periods), may be primary, associated with damage to the ovaries in childhood and adolescence, and secondary, arising in the reproductive period. In the first case of ovarian parenchyma growing organism sensitive to chronic infections (eg, tuberculosis), intestinal intoxication, eating disorders and other pathogens, resulting in the deaths observed some of the primordial follicles and development in their place, or fibrous connective tissue degeneration of the ovaries.

Depending on the degree of destruction of follicles ovarian estrogen excretion drops sharply, until the complete cessation.

In primary amenorrhea lack of estrogen production is accompanied by high levels of gonadotropins. Hypo- aestrogenemiya or influences the development of the sex glands and secondary sex characteristics. Such women are absent menstruation, observed infertility.

Secondary amenorrhea ovarian origin usually develops slowly, passing stage hypomenstrual syndrome. These studies colpocytologic in this case to determine, estrogen deficiency. Atrophy points to a sharp decline in estrogen production. Sufficient their products accompanied by certain proliferative changes in the vaginal epithelium, indicating the degree of safety function of ovarian follicles.

Uterine shape amenorrhea

Uterine shape amenorrhea develops due to dysfunction of the endometrium after traumatic manipulation (abortion, abortion, childbirth with septic complications) and in connection with specific chronic inflammation, especially tuberculosis, and etc.

Colpocytologic data in the form of uterine amenorrhea indicate preservation of the ovaries and the hypothalamic-pituitary processes, regulating their activities. Cytological changes reflect a sufficient degree of proliferation of the vaginal epithelium and often indicate a biphasic curve type colpocytologic.